If you reside in a house or apartment or simply need to ensure that your house is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the most straightforward is to limit your electronic device use. emf blockers could also consider EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your home against EMF radiation would be to install a RF shielding canopy. It is a type of net that contains EMF shielding and is used to block EMFs from entering a space. Another option is to get your house equipped with an electrical enclosure. They are referred to as Faraday cages.
A number of studies have proven studies have shown that the nonionizing EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation the cancer stem cell. This may account for the long-term responses seen in patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients is not clear.
The effects of AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumors were divided into three groups. One group did not have exposure RF EMF. The second group was exposed RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism of cancer-specific AM RF EMF could be based on the fact that cancer cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is mediated by CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have more broader implications in the treatment and diagnosis of various cancers.
The tumors in the control group were not exposed EMF from radiofrequency, and fed a standard mouse diet. The tumors of HCCMF HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours in the three groups also showed different growth curves. block emf treated with HCCMF saw a significant decrease in size of the tumor after eight weeks. However, the tumors that were treated using RCF showed no signs of shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is common in tumours that are exposed to RCF. The possibility is that the necrosis is caused by the lack of oxygen in the larger cancers.
In sum, the results indicate the fact that AM EMF has anti-cancer properties in vitro as well as in vivo. A number of studies have proven that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, emf blockers may exert a sustained impact on the development of HCC tumours in the vivo.